Wednesday, 11 June 2014

Description Of Some Of The Asanas For Blood Pressure

Relaxation in Corpse Position (Shavasana)
  • You may want to use a sweater, socks or a blanket to keep yourself warm during final relaxation
  • Lie on your back
  • Take a breath in and tense your whole body from head to toes: hold your breath, clench your fists, and squeeze all facial muscles as well as every single muscle in the body
  • Breathing out through the mouth with a “haaa” sound, release your muscles
  • Repeat one more time
  • Now lie in a position that feels comfortable to you. Keep the eyes closed
  • Mentally relax all parts of the body by taking your awareness to all parts of the body in turn, starting at the feet and ending with the head, feeling grateful for each part of the body. Gratefulness adds to the physical and mental relaxation
  • Let mother earth take all your weight, feeling the body to be very light. A relaxed body feels light
  • Next, allow your breath to relax by becoming aware of it and making it soft, small and quiet
  • Now allow your mind to relax by letting go of any worries, fears, anxiety or excitement. Surrender them all to God.For the time being, let go of any future plans or past events
  • Rest in the peaceful and blissful space within you
  • After resting for a few minutes, bring your awareness back to your body and take a couple of deep breaths
  • Gently roll over onto your right side
  • Slowly come up to the sitting position
  • Chant Om three times

Tuesday, 10 June 2014

INTER VERTEBRAL DISC PROLAPSE CASE TAKING

Place-trichy                                                                                   Date-11/06/2014


Pain
Radiculopathy
Severity
Chronicity
Numbness/burning/tingling and other neurological symptoms
Pain upon coughing/sneezing
Pain upon sitting/standing/walking
Wether pain present upon rest
Wether morning stiffness present or not
Any history of RTA/TRAUMA
Any history of LAMINECTOMY/DISCECTOMY
Any history of chronic physical strain
Any history of continuous travelling esp in two wheeler
Any history of sitting before computer
Other relevant occupational history

XRAY/MRI/NEURO REPORT/DOPPLER/CT SCAN ???

SLR-ZERO TO NINTY DEGREE, OR NEGATIVE

Gait, wether painful, spasmatic, trendelenberg, limping

Tenderness  +ve to ++++ve
Slight or mild tolerable discomfort on palpation
More severe pain on ordinary palpation, which the patient prefers not to tolerate
More intolerable pain even with light palpation or pressure
Pain which may be caused by even a mild stimulus such as a sheet touching the joint often characteristic of acute gout

Swelling +ve to ++++ve
Join swelling which may not be apparent on casual inspection, but should be recognizable to an experienced examiner
Joint swelling obvious even on casual observation
Markedly abnormal swelling
Joint swelling to a maximally abnormal degree

Limitation of motion 0 to 100%
About 25% loss of motion
About 50% loss of motion

About 75% loss of motion

100% loss of motion or complete ankyloses of the joint

DTR :
Biceps jerk-diminished, aggravated, absent, brisk, normal
Triceps jerk-
Knee jerk-
Ankle jerk-

Heel to toe walking-wether heel walking or toe walking more painful, or both normal or either of one painful.

Power of upper and lower limbs
·         Grade 0: No contraction or muscle movement
·         Grade 1: Trace of contraction, but no movement at the joint
·         Grade 2: Movement at the joint with gravity eliminated
·         Grade 3: Movement against gravity, but not against added resistance
·         Grade 4: Movement against external resistance, but less than normal
·         Grade 5: Normal strength

Modified Medical Research Council Scale for measuring hand muscles
Grade 5: full active range of motion & Normal muscle resistance
Grade 4: full active range of motion & Reduced muscle resistance
Grade 3: full active range of motion & No muscle resistance
Grade 2: Reduced active range of motion & No muscle resistance

Grade 1: No active range of motion & Palpable muscle contraction only
Grade 0: No active range of motion & No palpable muscle contraction
.......every information from the mouth of the patient is valuable, meant for beginners, thank you !



Monday, 9 June 2014

NOTES IN INFLAMMATION

Place : Thiruchirappalli at Aroggya Ayurveda Hospital
Date  : 09/06/2014

Inflammation is defined as the local response of living mammalian tissues to injury due to any agent. It is a body defense reaction in order to eliminate or limit the spread of injurious agents as well as to remove the necrosed cells and tissues.

CAUSES :
Physical agents like heat, cold, radiation, mechanical trauma
Chemical agents like organic and inorganic poisons
Infective agents like bacteria, viruses and other toxins
Immunologic agents like cell mediated and antigen antibody reactions

PROCESSES :
Early inflammatory response
Healing

SIGNS OF INFLAMMATION
Rubor
Tumor
Calor
Dolor
Functioleasa

TYPES OF INFLAMMATION
Acute &
Chronic

ACUTE INFLAMMATION
The changes in acute inflammation can be conviniently described under the following two headings :
Vascular Events
Cellular Events

VASCULAR EVENTS
Alteration in the micro vasculature(arterioles, cappillaries and venules) is the earliest response to tissue injury. These alterations include :
Haemodynamic changes
Changes in vascular permeability

HAEMODYNAMIC CHANGES
The earliest features of inflammatory response results from changes in the vascular flow and calibre of small blood vessels in the injured tissue.
Transient vaso constriction-3 to 5 seconds maximum 5 minutes
Persistent progressive vasodilation-mainly arterioles, 1/2 an hour of injury redness and warmth
Elevation of local hydrostatic pressure-transudation of fluid into ECF resulting in swelling
Slowing or stasis-due to increased permeability of microvasculature
Leucocytic margination-emigration

LEWIS EXPERIMENT
Features of haemodynamic changes. The triple response or Red line response :
Red Line-local vasodilation of cappillaries & venules
Flare-bright reddish appearance, vasodilation of adjacent arterioles
Wheal-swelling or oedema, transudation of fluid into the extravascular space

CHANGES IN VASCULAR PERMEABILITY
The appearance of inflammatory oedema due to increased vascular permebility of microvasculature bed is explained on the basis of starlings hypothesis. In normal circumstances, the fluid balance is maintained by two opposing forces :
-forces causing outward movement of fluid from microcirculation namely intravascular hydrostatic pressure and osmotic pressure of interstitial fluid
-forces causing inward movement of interstitial fluid into circulation namely intravascular osmotic pressure and hydrostatic pressure of interstitial fluid

MECHANISMS IF INCREASED VASCULAR PERMEABILITY
-Endothelial cell contraction, involving venules, immediate transient(15 to 30 min)response type, histamine, bradykinin others act as mediators, ex: during mild injury.
-Endothelial cell retraction, also involves venules, which is somewhat delayed(in 4 to 6 hrs) or prolonged (for 24 hrs or more), where IL-1, TNF act as mediators, happens in vitro only.
-Direct endothelial cell injury, where arterioles, venules, cappillaries are involved, immediate prolonged(hrs to days) or delayed prolonged(2-12 hrs, hrs to days), results in cell necrosis and detachment, ex : as in moderate to severe burns, severe bacterial infection, radiation injury.
-Leucucyte mediated endothelial injury,where venules and cappillaries are involved, which may be a delayed or prolonged process/response, where leucocyte activation release proteolytic enzymes, mostly happens in pulmonary venules & cappillaries.
-Neovascularisation, all levels are involved, which may be of any type, involving angiogenesis and vasculo endothelial growth factor usually seen in healing and tumors.

CELLULAR EVENTS
The cellular phase of inflammation consists of two processes
Exudation of leucucytes
Phagocytosis

EXUDATION OF LEUCOCYTES
Margination due to slowing and stasis results in exudation
Pavementing-neutrophil of central column come closer to vessel wall
Rolling of marginated and pavemented neutrophils
Transient bond between leucocytes and endothelial cells adhesion phase
Simultaneous to emigration of leucocytes,, escape of red cells through gaps between the endothelial cells , diapedesis takes place.
The chemotactic factor mediated transmigration of leucocytes after crossing several barriers to reach the interstitial tissues is called chemotaxis. Leukotriene B4 LTB4, Platelet factor 4 PF4, Complement system C3C5 in particular, Cytikines interleukins IL-1, IL-5, IL-6, Soluble bacterial products, Monocyte chemoattractant protein MCP-1, Chemotactic factor for CD4+T cells, Exotoxin chemotactic for eosinophils are the CHEMOKINES.

PHAGOCYTOSIS
Two main types of phagocytic cells
Polymorphonuclear neutrophils PMNs-microphages
Circulating monocytes-macrophages
Involves following four steps :
Recognition & attachment or opsonisation, IgG opsonins, C3b opsonins, Lectins.Opsonins coat microorganisms, occur naturally in serum
Engulfment stage
Degranulation stage
Killing or degradation stage

CHEMICAL MEDIATORS OF INFLAMMATION

CELL DERIVED MEDIATORS
Vasoactive amines like histamine , serotonin
Arachindonic acid metabolites, Prostaglandins PGs like PGD2, PGE2, PGF2 PGI2, TXA2 and Leukotrienes like LTB4, LTC4, LTD4, LTE4 etc...via the cyclo oxygenase pathway and lipo oxygenase pathway respectively.
Lysosomal components, granules of neutrophils, monocytes & tissue macrophages
Platelet Activating Factor
Cytokines which are polypeptide substances produced by activated lymphocytes and activated monocytes, namely IL-1, TNF alpha, TNF beta, IF alpha, IL 8, PF4, MCP 1
Nitirc oxide and oxygen derived derivatives

PLASMA DERIVED MEDIATORS
The kinin system
Clotting system
Fibrinolytic system
Complement system

FACTORS DETERMINING VARIATION IN INFLAMMATORY RESPONSE
Factors involving organisms
Factors involving host
Type of exudation
Cellular proliferation
Necrosis

MORPHOLOGY OF ACUTE INFLAMMATION
Pseudomembranous inflammation
Ulcer
Suppuration
Cellulitis
Bacterial infection of blood-Bactraemia, Septicaemia and Pyaemia : pyeamic abscess & septic infarcts.

SYSTEMIC EFFECTS
Fever
Leucocytosis
Lymphangitis-lymphadenitis
Shock

Fate of acute inflammation
Resolution
Healing of scarring
Progression to suppuration




Progression to chronic inflammation.

THANK YOU FOR SPENDING YOUR VALUABLE TIME READING. HAVE A GOOD DAY. GOD BLESS YOU.
 INITIATIVE TO SPREAD KNOWLEDGE OF PATHOLOGICAL PROCESS AMONG AYURVEDIC PEERS.
Dr.Prasanth.K.P. B.A.M.S.

INOCULATION INCEPTIVES FROM INDIA

English physician Jenner is credited with discovering vaccination on a scientific basis with his studies on small pox in 1796. Agroup of Fellow of Royal Society had earlier studied this method of inoculating people in the 1760s. Dr.J.Z.Howell , one of the members who was in the Bengal province formore than 10 years to study the Indian Vaccination method, lectured at the London Royal College of Physicians in 1767 "that nearly the same salutory method, now so happily persuaded in England,.......has the sanction of remotest antiquity(in India) illustrating the propriety of present practise."

Dr.J.Z.Howell writes the most detailed account for the college of Physicians in London in 1767(An account of the manner of inoculating for the small pox in the East Indies, by J.Z.Howells, F.R.S. addressed to the president and members of the college of physicians in London. He wrote,
ions
"Inoculation is performed in Indostan by a particular tribe of Brahmins, who are delegated annually for this service from the different colleges of Bindoobund, Elaabas and Benares, over all the distant provinces, divide them into small parties, of three or four each, they plan their travelling circuit in such wise as to arrive at the places of the operation consists only in abstaining for a month from fish, milk and ghee.When the brahmins begin to inoculate any who have not , on a strict scrutiny, duly observed the preparatory course enjoined in them. It is no uncommon thing for them to ask the parents how many pocks they choose their children should have."

On the efficiency of this practise Howell has the following to say :

"when the before recited treatment of the inoculation is strictly followed, it is next to a miracle to hear, that one in a million fails to recieving the infection, or of one that miscarries under it."

The sactya grantham-ancient brahman medical text 3,500 years old describing brain surgery and anasthetics, contains the following passages giving instruction on small pox vaccination.

"Take on the tip of a knife the contents of the inflammation, inject it into the arm of the man, mixing, it with his blood. A fever will follow but the malady will pass very easily and will create no complications."

Two of the more important medical arts of India - plastic surgery and inoculations against small pox. Both these were indigenously evolved and the accounts we have, come from Westerners sent out to study them. One of those curious facts was the inoculation against small pox disease, practised in both north and south india till it was banned or  disrupted by the English authorities in 1802-03. The ban was pronounced on ''humanitarian " grounds by Superintendent General of Vaccine.

Sunday, 1 June 2014

BIPOLAR DISORDER

Bipolar disorder by definition is a mental illness characterised by episodes of an elevated mood known as mania, usually alternating with episodes of depression.
During mania an individual feels abnormally happy, energetic, or irritable , but often makes poor decisions due to unrealistic ideas or poor regard of consequences.
Maniac and depressive episodes can impair the individuals ability to function in ordinary life.
SIGNS & SYMPTOMS.
1.Mania
2.Hypomania
3.Psychosis
Maniac episodes.
-distinct period of atleast one week of elevated or irritable mood which can take the form of euphoria & exhibit three or more of following behaviours :
-speak in a rapid uninterrupted manner
-easily distracted
-having racing thoughts
-display an increase in goal oriented activities or feel agitated
-exhibit behaviours characterised as impulsive
-excessive money spending
-to meet criteria for mania episodes these behaviour must impair the individuals ability to socialize or work.
IF UNTREATED A MANIAC EPISODE LAST 3-6 MONTHS
Hypomaniac episodes
-milder form of mania defined as four days of the same criteria for mania with lesser symptoms
-does not cause a significant decrease in individuals ability to socialise or work ,lacks psychotic features and does not require psychiatric hospitalisation
-overall functioning may increase during episodes of hypomania and is thought as a defence mechanism against depression
-hypomaniac episodes rerely progress to true maniac episodes
-patient may feel good even when family and friends recognise mood swings
Depressive episodes
-persistent feeling of sadness
-anxiety
-guilt
-anger
-isolation
-hopelessness
-disturbances in sleep and appetite
-fatigue and loss of interest in usually enjoyable activities
-problems concentrating
-self loathing
-apathy or indifference
-depersonalisation
-loss of libido
-shyness or social anxiety
-irritability
-chronic idiopathic pain
-lack of motivation
-morbid suicidal thoughts
-severe cases psychotic delusion & hallucination
Mixed affective episodes
-high risk for suicidal behaviour
-hopelessness often paired with mood swings or diificulties with impulse control
-anxietty disorder occur more frequently as a co morbid condition
Associated features
-changes in cognitive processess & abilities
-reduced attentional capabilities
-impaired memory
-diificulty in maintaining relationships
Comorbid conditions
OCD/SUBSTANCE ABUSE/EATING DISORDERS/ADHD/SOCIAL PHOBIA/PMS/PANIC DISORDERS
CAUSES
Genetic
-60 to 80 % account for genetic influences
-risk of bipolar disorder is ten fold higher in 1st degree relatives of those affected with bipolar disorder when compared to general population
Physiological
-increase in volume of lateral ventricles, globus pallidus, and increase in rates of deep white matter hyperintensities
-abnormal modulation between ventral prefrontal and limbic regions especially the amygdala
-early life stress dysfunction of hypothalamic pitutary adrenal axis leading to its over activation
-alterations to mitochondria and sodium ATP ase pump are believed to cause cyclical periods of poor neuron firing(depression) and hypersensitivity neuron firing(mania)
Environmental
-traumatic abusive episodes in childhood
-stressful events in childhood
Neurological
-stroke
-traumatic brain injury
-multiple sclerosis
-porphyria
-temporal lobe epilepsy
Neuroendocrinal
-dopamine
-gamma amino butyric acid
-glutamate, these are neuro transmitters known for mood cycling.
Evolutionary origin of bipolar disorder states that during short summers of extreme climatic zones , hypomania would be adaptive, allowing completion of many tasks necessary for survival within a short period of time. During long winters the lethargy , hypersomnia, lack of interest in social activities, and over eating of depression would be adaptive to group cohesion & survival.
Diagnosis
M-severe mania
D-severe depression
m-less severe mania
d-less severe depression
BP 1 Disorder                   : Md
BP 11 Disorder                 : mD
Cyclothymia                      : md
Non Specific BP Disorder  :  any symptomsM/D/m/d
Differential Diagnosis
-schizophrenia
-major depressive disorder
-attention deficit hyperactivity disorder
-borderline personality disorder
Medications
-cognitive behavioural therapy
-family focussed therapy
-psycho education
-interpersonal & social rythm therapy
-anticonvulsants,antipsychotics
AYURVEDIC APPROACH TOWARDS BIPOLAR DEPRESSIVE PSYCHOSIS
Ayurveda offers a multitude of holistic tools and ways of approaching this disease that can restore health so as to not rely as heavily on conventional treatments.
Ayurveda uses a general term UNMADA for insanity. According to Charaka unmada is wandering about of mind, intellect, consciousness, knowledge, memory,, inclination, manners, activities and conduct.
According to Ayurveda bipolar disorder manifests due to imbalance in three fundemental biological qualities that govern body and mind ; vata, pitta and kapha and the mental principles of sattwa, raja, tamas, which are the qualities of consciousness.
Some modern ayurvedic practitioners like BPD to lack of stability of ojas. OJAS is defined to maintain immunity, strength, integrity and vitality. It acts as containers that holds the other two energies in the body ; tejas-the energy of intelligence and discrimination; and prana the body’s life energy. During elated phase , praana and tejas are high, during depressive phase will be low and tejas can be either high or low.
The maniac , depressive and hypomaniac symptoms can be caused due to vata, pitta, kapha or two combinations of these and all the three combinations of the dosas.
When vata causes elation symptoms such as talkativeness, excessiveness, racing thoughts, hyper sexuality, euphoria, impulsive over spending are seen.
When vata causes depressive symptoms emptiness, hopelessness, difficulty concentrating, insomnia, restlessness, memory problems, uncontrollable crying, weight loss, despair are seen.
When pitta causes elated symptoms over-confidence, irritability, aggression. anger, under-sleeping, hyper activity are seen.
When kapha causes depressive symptoms feelings of sadness, lethargy, lack of enthusiasm, apathy, lack of appetite are seen.
AYURVEDIC TREATMENTS
-regular solid routines and stability, regular waking and sleeping, regular meditation and exercise in morning
-identify and minimise the stressors that trigger changes, reduce workload, keep home tidy, clutter free and clean, give time for everything, it is best healer
-avoid stimulants like coffee and beverages
-nervine tonics become the most important for building stability and mental ojas
-common herbs used are jyotismathi, gingko, jatamansi, oat straw, valerian root, st,john’s wort, gotu kola, aswagandha, brahmi, shatavari, ginseng, shanka pushpi, nutmeg, skull cap, kapikachchu, haritaki, bhrungaraj
-clours like yellow, green, gold, blue, white, violet and purple are considered sattvic colours
-sandalwood, jatamansi, rosemary, cinnamon, thyme, mint, eucalyptus, rose, basil, lavender
-exercise but no over exertion, yoga, qi, gory, pilates and tai chi, aikido, gardening, walking, gentle hiking
-PANCHA KARMA :
 1.therapeutic purgation VIRECHANA in maniac phase
2.therapeutic enema BASTI for vata alleiviation
3.therapeutic emesis VAMANA in depressive phase
4.oleation or SNEHA PRAYOGA for palliation
5.SIRODHARA & SIROBASTI for direct action over brain, mind and subtle energy fields.
Ayurveda is not a quick process and relies on development of healthy routines and habits over a life time, it is important for a patient already diagnosed as bipolar to continue with treatment laid out by his or her physician.
AYURVEDA THE COMPLETE REMEDY, THE COMPLETE CURE !!!


Wednesday, 28 May 2014

Pune doc finds Ayurvedic kidney stone treatment

Urologist’s Research to be presented in US

  • DNA RNA Research & Archives

Pune: City-based urologist Dr Suresh Patankar has conducted a research on ‘Kidney Stone and Wound Healing’, which he will present at the American Urological Conference at Orlando City, USA on May 17, addressing about 10,000 urologists from the world over. The research is now in the final stages. It includes a study on treating small kidney stones, detecting them at the early stage and reducing chances of their recurrence.
Dr Patankar, who is also the founder of Ace Hospital and Research Centre, said the research is based on Ayurvedic concepts but has been being conducted using modern scientific methods. He believes this will change the outlook of modern practitioners towards Ayurveda.
He said kidney stone in urology, and wound healing in all surgical procedures are common problems and need to be studied in detail. “A lot of new surgical treatments are available to remove kidney stones with minimum damage and pain to the patient. These treatments are being carried out by our hospital for the last 30 years. However, we have observed that for small-sized stones no satisfactory treatment is available. We are working on that,” he said. All small stones eventually become large and require medical attention. Also, kidney stones are a recurring problem. Dr Patankar and his team are also researching methods of treating kidney stone in the early stages.
The second part of the research concentrates on wound healing. In certain surgeries, if the wound does not heal properly, it may lead to complications in the surgery or to its failure. Dr Patankar has found a regenerative formula, which promotes proper and faster wound healing with less scaring. This research has been accepted by the scientific community after clinical studies were conducted on human beings.

THE FIVE DAYS IN THE HOT SEAT !!!

आयू  कामय  मानेन  धर्मार्थ  सुख  साधनम्  
आयुर्वेदोपदेषेषु  विधेय  परमादर 


It was decided earlier, his vacation (of the chief doctor), who happens to be a veteran in treating Back pain cases here in Trichy. It has happened before and without saying anything he usually asks me to take his place. Every time he gives some challenges and raises the bar, so you gotta learn everytime to be in his good books. This time around he put forward a rather "invincible" task of admitting 10 in-patients. 10 in 5, admitting patients in an ayurvedic hospital is a "coaxing'', given that unlike allopathy, ayurvedic treatments are optional, they could manage with pain killers ! But only when they reach that cliff of going for a surgery( these patients bargain with their health ! ) OR no relief with pain killers that they opt for Ayurvedic treatments. Couln't you treat it for less ? Is your health of that much value to you ? You are able to satisfy your logic in spending LAKHS for surgey to end up in disdain, but spending 1/3 rd of that amount for a long lasting relief life changing palliative treatment looks less attractive to you ! Everything in our life comes at a cost other than GODS GRACE ! You do good or bad god treats you equally, he gives equal oppurtunities, only thing is wether you realise or not !

I have to admit patients, convince them, explain them, and treat to show them results. But this time around due to my previous long interaction with in-patients, consultation was less fussy. Given that I had enough of know-how to treat, I had some nervousness that shown by a beginner, it can become devastating, the patient stares into your eye as if for help and even the slightest of negativity in your approach sends opposite signals, so you gotta be steady, stable, cheerful. When you take the profession seriously, constantly interact with the seniors in the field, gather knowledge, update yourself, keep on trying, then everything falls into place, you dont even have to plan or think or try. A handful of cases to consult was an enriching experience in itself , useful for a life time ! I got a brief oppurtunity and I was prepared, hope oneday I will stand on my own !!!